Wednesday, November 5, 2014

Theory 1A cell filtering diagram

This construct allows for an absence of potentially harmful membraneous filters, instead relying on the cell markers' own polarity to draw them from the stream, allowing for a far more fluid deconstruction, and replacement.
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Eternal Youth by Matthew Lane is licensed under a Creative Commons Attribution-NoDerivatives 4.0 International License.

Thursday, October 23, 2014

Theory 1A addendum 5

Eternal Youth, utilizing the device, can be refined to a more accurate form than simple regulation of cell number, by adding a wireless connection that its findings can be reported, such that an influx of white blood cells is reported to the corresponding clinic, and the patient can be alerted and treated.
Addition of a second duct, to draw the aging cells into a specialized chamber, and disassembled with chemicals garnered from white blood cells, or by covering them with antigens, and releasing white blood cells, allows for a replenishment of spent cellular components,  as several filtering membranes can differentiate the fragments by size and structure due to its 1) porosity, and 2) chemical layering.
As cell levels will fluctuate with illness or age, the device must be programmed to report this, and the staff receiving this, can dismiss it as related to a harmless growth or a missing follicle. 
Macrophages and Stem cells are released intermittently, to account for aging of tissue that may not reach the device’s sensors, relying on the natural degradation of cells, and divulging sufficient stem cells to replace those that division cannot, and are guided into place using chemical signals to urge it into motion, as observed in normal expression during cellular motion. 
Sequestered from the remainder of the device, is a section containing live pig bladder components, which are stripped for their matrix, which expresses regenerative properties, and is thus guided into place using cells equipped with filamentous limbs, such that should tissue be damaged and detected due to blood loss, regeneration can be incited, allowing for internal restoration of aged organs and tissue, altering an elderly body into a youthful form.
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Theory 1A addendum 5 by Matthew Lane is licensed under a Creative Commons Attribution-NoDerivatives 4.0 International License.

Thursday, October 9, 2014

Diagrams for previous addendum

Foreword: I have in the past, and may in the future neglect to name the cell properly. Some cells have a plasma membrane rather than a wall, yet I refer to the cell's border, be it composed of a membrane or wall, and thus the concept remains whether its words fail to properly define.
These diagrams are intended for the simplification of otherwise confusing subjects, and are protected by creative commons:
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Tuesday, October 7, 2014

Theory 1A Addendum 4

The marker(perhaps hewn of peptides so as to avoid bodily damage, and a polar molecule to be charged, its dipole slowly drawing it toward the receptor on the cell surface, at the same rate the telomere degrades), is contained within the nucleus and rises to the cell surface to be read by the programmed device, by: its charge is opposite that of the ambient fluid, and the telomere, that it is held in limbo until one force is reduced such that it is released 1) or it is composed of materials that will react with neither substance, and rises to be recorded, by using thin strands to grasp the telomere, that once it shortens, the marker is freed, and floats about, and is drawn to the surface by the charge of its tendrils. Yet this only accounts for the blood, thus the organs and tissues are rejuvenated by sending forth a stream of macrophages into the body, to clear dead cells, or the second and more likely possibility of using cells engineered with a receptor that draws them to an antigen like structure released upon necrosis or apoptosis, attached by similar means as the marker. This cell then consumes the dead peer, then depending on the type of structure the dead cell bore i.e marrow, brain… the cell releases a second marker to its surface, from several it houses within a reprogrammed organelle, released by the type of marker it consumed reacting to the marker within itself, drawing it forth. Cycling through the body, its passage through the device releases a cell of equivalent type to that which perished, which is then guided into place by chemical signals from the implant, sending the cell through the veins, and to its location, then creating a tear much like that of some blood vessels, which are of meager size and thus allow only one cell to escape. This completed, the device waits several moments, for the cell to depart, then fuses the vein. Should the organ or tissue not require this, such a process is not initiated, and if multiple cells depart for the same location, it will allow the gap to remain open, yet this is a parlous endeavor, and thus for surface tissue to be healed and rejuvenated, a second implant is required, entrenched within the flesh, yet capable of directly impacting the skin. It sends forth a permeable membrane, that is guided by its schematics of the body garnered from a mapping prior to entry, by those implanting the device, sending a probe in the form of an engineered cell through the body, and using it to create an internal map. This guides the membrane, upon which a new layer of flesh forms, prepared to replace any damaged surface tissue by simply rising upward. This replenishes surface tissue, and a second layer beneath may be used to restore muscle and bone, or those far less swiftly accessed by veins. 
For the device to find success, it must be capable of issuing forth streams of cells sufficient to replace those slain by age, without exception, and thus the algorithm for dissemination of cells from its storage, must utilize an exact calculation of the cells not replaced by division, and account for this to retain healthy cell levels as the body ages, for as apoptosis is required for some processes, the cells become weary and incapable of holding erect the human vessel they have for so long inhabited, and thus must be replaced. This rate can be gathered by analyzing a sample, and mathematical extrapolation thereupon.

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Eternal Youth by Matthew Lane is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.
 

Friday, October 3, 2014

Theory 1A Addendum 3

Using a programmed device, rather than the marrow cube, allows for more accurate, and preferable results: current attempts at engineering T-cells to target cancer have been largely effective in targeting the cancer’s peptides, thus the device’ breeding chambers would be filled with these as well, release of these into the body evaded by a thin membrane whose pores allow cells to pass forth, yet thin arms extending from these cells do not allow them to escape, and the elasticity of this membrane simply urges them back into the chamber.  This device could detect the number of cells in the body by marking those that have already passed with a harmless capsule on the surface, that will be dissolved once the total number has been counted, by a T-cell programmed to detect this marker, yet rather than destroy or consume the cell, it releases the dissolving agent for the marker, removing it. This device could more accurately account for aging, and cell reduction in addition to using specially engineered cells armed with thin limbs that latch onto a cell, and move it into position, guided by a receptor on their surface, that detects chemical signals issued forth by the device, allowing for a swifter, and more accurate replacement of cells that have succumbed to age.
The device avoids clotting the blood, or disrupting ordinary flow of blood, by being housed within a small chamber of bone inserted into the chest, or shoulder. Leading to it, are several small veins of engineered cells, closed by a sphincter at the electric signal of this device, allowing it to isolate itself if so desired.

Theory 1A Addendum 2

With such a form of eternal youth, the current method of immune system reprogramming may be used to halt cancerous cells from exiting the ducts, yet this may be unnecessary: the amount of cdc4 and cell division proteins cast into the respective chambers is minuscule, sufficient only to replace the cells released, and little more. The white blood cells and macrophages in both chambers will prevent the release of dead cells into the body, and unregulated growth may trigger a lining on the walls of the chamber, unnatural numbers of cells grating against it dissolving the capsules thereupon, and releasing sufficient antigens to destroy all cells within, then the virus draws additional cells, or more can be implanted that the process will continue, yet the amount of cdc4 will change, tempered to avoid carcinogenic cells in the future.

Monday, September 29, 2014

Theory 1A Addendum

By using a shell of one of the enzymes dissolved by the nucleoplasm, the virus can enter the cell, and penetrate to the telomere, then the virus either retreats or perishes, and the marker it leaves on the telomere, to avoid chemically altering the telomere by embedding the marker, its polarity holds it at gentle repulsion from both nucleoplasm and telomere, its molecule serving little purpose yet possibly housed within a polarized capsule to hold it in stasis until the telomere is dissolved by age, releasing this marker from the cell as the repulsion of the nucleoplasm forces it outward, possibly releasing the antigens within which can force consumption of this dying or dead cell, yet this would involve a secondary virus attracted to this marker, to change the white blood cell into a stem cell, thus 2) the marker may attract a virus cell or rather an infected macrophage or white blood cell, that upon consumption, the marker enters its cytoplasm, dissolves, and in so doing releases a second capsule which is formed within a vacuole or specialized organelle(possibly an edit of the Golgi apparatus, installed by the virus, that compacts the genes of stem cells into a small packet and cell division proteins into another, which is then released into the marrow cube, as the infected cell passes through, the capsule being polarized opposite to the membrane, and thus slamming into it to cause the change in current, and opening of the muscle tissue or sphincters on the stem cell/macrophage ducts of the cube/orb, the membrane's 1) hinge like motion releasing the capsule into the stem cell breeding chamber, and the proteins into the macrophage breeding center, or 2) the enzyme shell on the capsule dissolves, and the enzymes slide along the membrane, and into the chambers.